and John Dennison2
(1)
Department of Psychological Medicine, University of Otago School of Medicine, Wellington, New Zealand
(2)
Department of Anatomy, Otago Medical School, Dunedin, New Zealand
Course of chronic psychoses
Aetiological classification
Griesinger’s primordial deliria
Lecture
Gentlemen!
With the summary knowledge of paranoid states that you have acquired so far, I realize that so far you have become acquainted only with certain dominating symptoms; but many others that occur concurrently can also be found only as acute psychoses. However, you will now be able to discover the main features of the vast majority of paranoid states—and so satisfy the requirement which must be fulfilled, with examples, in each psychiatric report, namely that psychotic symptoms which constitute the mental disorder in each specific case must be recorded in precise detail—a requirement that sadly is seldom met in reports of even so-called authorities. I cannot emphasize strongly enough that you have the right to declare a person mentally ill only when you can produce evidence of this by establishing definite psychotic symptoms; only then will you be spared the embarrassment of your opinion being exposed to justified attacks by lay people. The ‘general impression’ [W] sometimes relied on even by better known representatives of our profession, when they fail to elicit definite psychotic symptoms, is no better than everyday parlance and must elicit the deepest suspicion, when used as the basis of diagnosis of a paranoid state. It deserves to be rejected most strongly when, in such cases, the claim is made that we are dealing with a well-known and relatively simple disease state, given the accurate name of paranoia chronica simplex. Then, it is easy to arouse the impression of intentional deception, for both judges and lay people, thereby harming the reputation of the entire alienist profession. If you want to avoid such mistakes, please note that the most obvious self-aggrandizement is far from any grandiose delusion; mistrust and hatred of a few—or many—people is not a persecutory delusion; and paranoid states are mental disorders which are always relatively easy to grasp, detected by very specific psychotic symptoms.
In some of our most widely read textbooks you will see sub-classification of paranoia into paranoia chronica simplex and hallucinatoria. You can infer from my comments how much or rather how little such distinctions are justified. There is no merit at all in independent terminology for hallucinations, at least for phonemes, which almost always predominate.
Perhaps it requires a certain apology, when, with motives such as I have just outlined, I choose the term ‘paranoid states’ [W] to cover all chronic mental disorders in which material alterations of consciousness are dominant. One might object by demanding that I at least make an attempt to delineate clearly the supposedly well-known paranoia chronica simplex [W]. I deliberately avoid such an attempt because, in my opinion, there is no well-known illness of this type, unless you want the name to monopolise that very small number of individual cases that happen to match one another. I come back to those cases shortly (p. 102).
With regard to the broader range of paranoid states, our lack of our knowledge is particularly noticeable when we try to subdivide them according to their respective course and later development. In this regard, we are familiar with only some limiting cases. I shall briefly recapitulate these here, according to data that I have often touched on. Among residual mental disorders are some we have come to recognize as stable states, usually characterized by low incidence of symptoms, and integrity of other functions. Then, there are other cases, about which we know little, which are characterized by very gradual, creeping development, and an equally slow, steadily progressing course, never amounting to actual acute attacks or severe exacerbations. For both types of illness, as for all major changes, we can construct a disease curve corresponding to the expansion of the disease state. In the first situation the corresponding curve remains parallel with the x-axis; in the second—it rises uniformly and slowly away from the x-axis.
A few remarks are directed to those latter cases with an extremely chronic course. They correspond to the rather more common type, of slowly emerging persecutory delusions, which, after some time, may evolve into grandiose ones. The period of slow, imperceptible development in such cases, which often makes it difficult to determine the precise starting date, is characterized by occurrence of delusions of relatedness, and, shortly afterward, by isolated phonemes with the same content. An overvalued idea may take hold as the first and foremost symptom, and determine the substance of delusions of reference. Gradually the phonemes get out of hand, and other sensory hallucinations and abnormal sensations may even join in. Persecutory ideas are then systematized in two ways. One is via delusions of explanation, which refer to the hallucinations themselves: the so-called physical persecution complex [Ed], with hallucinations related in turn to the agent behind the persecution, and his motives. Grandiosity preferentially links up with a series of explanatory delusions, because of the logical necessity of explaining how such a large following—an array of so many people—should develop. If discernment and formal thought processes are well preserved, this finally leads to formation of technical terms of a more or less adventurous nature, often appearing at first as phonemes, providing patients with newly formed words. Such patients express themselves in a changed manner, which is very characteristic. By means of their intact sense of logic, their entire world view is gradually transformed and, depending on the sick person’s state of mind, philosophical systems may be advocated with quite outlandish structures. Formally correct logic and unmistakable intellectual productivity remain right to the end. In the philosophical literature of the last decade, you may have been struck by a multi-volume work written by an apparently mentally ill scholar, which would have left you shaking your head. The final outcome of the full disease process is a material change in all three domains of awareness, because the patient sooner or later becomes aware of physical changes in his body. Thus, if you want to choose a name for such cases, then in the later stages you are dealing with ‘chronic total psychosis’ [Ed]. The initial symptoms are likewise beyond any question, the entire first period of the illness manifest predominantly in the allopsychic area. Delusions of relatedness are detectable by themselves for a long time, entirely of an allopsychic character as just described. Hallucinations with explanatory delusions bring about gradual, but inexorable, reinterpretation of the outside world; and for initial years of illness, the designation of ‘chronic worsening allopsychosis’ [Ed]—also ‘chronic hallucinosis’ [Ed]—is justified. The qualifier ‘worsening’ [Ed] indicates that the psychosis gradually becomes total, since, from the time of onset of grandiose delusions, it also encompasses the autopsychic domain. That it never comes to more severe symptoms in the area of motility also seems characteristic of such cases. While relatively common (pp. 63, 64), only a small absolute number belong to the class of ‘purely chronically progressing’ [Ed] cases. These correspond most closely to the commonly alleged paranoia chronica [W]. The two female patients, Schmidt and Reising, are examples of this—the latter, right from the outset, in conjunction with an overvalued idea.
A third trajectory for paranoid states has become known to me over the course of time through several cases. It develops in a special manner, representing continuation of a bout of acute psychosis, after surviving several such bouts in previous years with total recovery. Up to the present, I have seen such a disease process after acute hallucinosis, or acute hallucinatory allopsychosis (see later), usually in alcoholics. Progression of the chronic condition, which always takes the form of a physical delusion of persecution, here seems to be more rapid and deleterious than for the aforementioned chronic forms.
I can mention a fourth form of paranoid state only in anticipation here. Amongst acute psychoses, about which we will learn later, is the state of depressive melancholia, an acute general illness characterized by intermingling of Affect and general akinesia of intrapsychic origin. Such a melancholic state can, for a long time, imply existence of pure melancholia, until clues are provided from a patient’s altered behaviour that intrapsychic dysfunction is subsiding, but at the same time, delusions of relatedness and corresponding phonemes appear. Usually it is any outbreak of indignation that prompts a patient to speak out, and with one stroke, to reveal an entirely different picture. The paranoid state, which is now revealed, is usually made up of persecutory and grandiose delusions in equal measure. Outbursts of wild ranting and a tendency to violence accompany it in nearly characteristic ways, leading quite soon to falsification of the entire contents of consciousness, to the point of material confusion. Patients seem to be able to remain in this stage of major confusion for a long time, although with decreasing energy in their actions, but without actual dementia. I am not yet clear of the final outcome. To avoid misunderstanding, I notice that the substantial confusion is by no means due exclusively to this course of events, but occurs with any extensive falsification of consciousness. Patients ultimately function using a series of idiosyncratic terms, making them unintelligible to other individuals, and they may even be understood falsely by others. Such confusion is then only apparent confusion, not noticed at all by another individual with just the same falsifications of consciousness.

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